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Author: Tom G Richardson

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    • bioinformatics: 921 (rank: 10,309 (tie) out of 28,921)
    • epidemiology: 2,035 (rank: 500 out of 9,537)
    • genetics: 10,428 (rank: 1,083 (tie) out of 32,490)
    • genomics: 689 (rank: 19,906 (tie) out of 36,992)

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Preprints

Phenome-wide Mendelian randomization mapping the influence of the plasma proteome on complex diseases

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An atlas of polygenic risk score associations to highlight putative causal relationships across the human phenome

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A transcriptome-wide Mendelian randomization study to uncover tissue-dependent regulatory mechanisms across the human phenome

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Systematic Mendelian randomization framework elucidates hundreds of genetic loci which may influence disease through changes in DNA methylation levels

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Causal epigenome-wide association study identifies CpG sites that influence cardiovascular disease risk

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PhenoSpD: an integrated toolkit for phenotypic correlation estimation and multiple testing correction using GWAS summary statistics

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Identification of loci where DNA methylation potentially mediates genetic risk of type 1 diabetes

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Using Y chromosomal haplogroups in genetic association studies and suggested implications

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Integrating Mendelian randomization and multiple-trait colocalization to uncover cell-specific inflammatory drivers of autoimmune and atopic disease

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Prioritizing putative influential genes in early life cardiovascular disease susceptibility by applying tissue-specific Mendelian randomization

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Investigating causal pathways between liability to ADHD and substance use, and liability to substance use and ADHD risk, using Mendelian randomization.

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Conditioning on a collider may induce spurious associations: Do the results of Gale et al. (2017) support a protective effect of neuroticism in population sub-groups?

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DNA methylation links prenatal smoking exposure to later life health outcomes in offspring

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Leveraging brain cortex-derived molecular data to elucidate epigenetic and transcriptomic drivers of neurological function and disease

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Genetic predictors of participation in optional components of UK Biobank

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DNA methylation mediates genetic liability to non-syndromic cleft lip/palate

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