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Author: David A. Hinds

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  • All-time downloads: 43,036 (rank: 2,699 out of 509,618)
  • Categories:
    • genetics: 39,619 (rank: 104 out of 38,578)
    • genomics: 3,417 (rank: 6,417 (tie) out of 45,500)

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Preprints

Expanding Parkinson's disease genetics: novel risk loci, genomic context, causal insights and heritable risk.

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Genome-wide Analysis of Insomnia (N=1,331,010) Identifies Novel Loci and Functional Pathways

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Genome-wide meta-analysis of depression identifies 102 independent variants and highlights the importance of the prefrontal brain regions.

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Genome-wide association analyses of risk tolerance and risky behaviors in over 1 million individuals identify hundreds of loci and shared genetic influences

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Genetic analyses identify widespread sex-differential participation bias

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Genome-wide association study identifies 48 common genetic variants associated with handedness

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Genome-wide association analyses of chronotype in 697,828 individuals provides new insights into circadian rhythms in humans and links to disease

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Genome-wide analyses of self-reported empathy: correlations with autism, schizophrenia, and anorexia nervosa

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Genetic variants associated with motion sickness point to roles for inner ear development, neurological processes, and glucose homeostasis

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Genetic Associations with Subjective Well-Being Also Implicate Depression and Neuroticism

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Genome-wide association analyses in >119,000 individuals identifies thirteen morningness and two sleep duration loci

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Genome-wide association and HLA region fine-mapping studies identify susceptibility loci for multiple common infections

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An Atlas of Human and Murine Genetic Influences on Osteoporosis

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Genome-wide meta-analysis of cognitive empathy: heritability, and correlates with sex, neuropsychiatric conditions and brain anatomy

genetics more details view paper
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Genome-wide association studies of impulsive personality traits (BIS-11 and UPPSP) and drug experimentation in up to 22,861 adult research participants

genetics more details view paper
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Social and non-social autism symptom and trait domains are genetically dissociable

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